Prioritizing genes responsible for host resistance to influenza using network approaches

Keyphrases

• Network Approach 100%
• Influenza 100%
• Host Resistance 100%
• Quantitative Trait Loci 66%
• Influenza-like Illness 66%
• Resistance Genes 66%
• Cutout 33%
• Tumor Necrosis Factor-α 33%
• Signaling Pathway 33%
• Complexity Issues 33%
• Human Subjects 33%
• Pro-survival 33%
• Molecular Mechanism 33%
• Novel Mechanism 33%
• Two-pathway 33%
• Survival Effect 33%
• Mouse Strain 33%
• Pro-apoptotic 33%
• Apoptotic Effect 33%
• Expression Profile 33%
• Genetic Association 33%
• Gene Expression Analysis 33%
• Lines of Evidence 33%
• Partial Knowledge 33%
• Novel Pathways 33%
• Moral Dilemmas 33%
• Complex Traits 33%
• Functional Investigation 33%
• Inbred Mice 33%
• Host Condition 33%
• RNA Interference Screen 33%
• Genetic Makeup 33%
• Network-based Methods 33%
• Delicate Balance 33%
• Apoptosis-necrosis 33%
• TNF Receptor 2 (TNFR2) 33%

Biochemistry, Genetics and Molecular Biology

• Host Resistance 100%
• Quantitative Trait Locus 66%
• Influenza Virus 66%
• Tumor Necrosis Factor 66%
• Candidate Gene 33%
• Gene Expression Profiling 33%
• Genetics 33%
• Genetic Association 33%
• Mouse Strain 33%
• Inbred Mouse Strain 33%
• Tumor Necrosis Factor Receptor 33%
• RNAI 33%
• Homo sapiens 33%
• Programmed Cell Death 33%