A schizophrenia-related sensorimotor deficit links α3-containing GABA_Areceptors to a dopamine hyperfunction

Keyphrases

• Schizophrenia 100%
• Dopamine 100%
• GABAA Receptor (GABAAR) 100%
• Hyperfunctions 100%
• Sensorimotor Deficits 100%
• Knock-in Mouse Model 66%
• Psychiatric Conditions 33%
• In(III) 33%
• Information Processing 16%
• Haloperidol 16%
• Symptomatology 16%
• Prepulse Inhibition 16%
• Spontaneous Locomotor Activity 16%
• Acoustic Startle Reflex 16%
• Sensorimotor Gating 16%
• Overactivity 16%
• Antipsychotics 16%
• Effective Treatment 16%
• Amphetamine-induced Hyperlocomotion 16%
• Developmental Defects 16%
• Sensorimotor Information 16%
• WT Mice 16%
• Diazepam 16%
• D2 Receptor Antagonists 16%
• Dopamine Function 16%
• Elevated Plus Maze Test 16%
• Midbrain Dopaminergic Neurons 16%
• Anxiolytic Activity 16%
• Brain Abnormalities 16%
• Dopaminergic System 16%
• Sensorimotor Gating Deficit 16%
• Anxiety-like Behavior 16%
• Mouse Pointing 16%
• GABAergic 16%
• GABAA Receptor Subunits 16%
• Whole-cell Currents 16%

Neuroscience

• Mental Disorder 100%
• Sensory Gating 100%
• Dopaminergic 50%
• Locomotor Activity 50%
• Brain Abnormalities 50%
• Behavior (Neuroscience) 50%
• Information Processing 50%
• Midbrain 50%
• Elevated Plus Maze 50%
• Agonist 50%
• Startle Response 50%
• Receptor Subunit 50%
• Gamma-Aminobutyric Acid 50%
• GABAA Receptor 50%
• Receptor Antagonist 50%
• Prepulse Inhibition 50%
• Haloperidol 50%
• Diazepam 50%
• Amphetamine 50%
• Antipsychotic 50%